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In many countries, glutathione is permitted as a cosmetic ingredient provided it does not make therapeutic claims
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Mitochondrial dysfunction and excessive production of reactive oxygen species promote inflammation and immune dysregulation in SLE
The body is unable to remove them efficiently, so they accumulate as biological waste that disrupts tissue homeostasis and accelerates degenerative changes
Its interaction with interleukin-1 (IL-1) plays a role in dampening the inflammatory response in the central nervous system, which is pivotal in mitigating neuroinflammation
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