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Hu H, et al

These multiple cellular pathways explain why GHK-Cu attracts attention, but they also make it harder to reduce the peptide to one simple mechanism of action

supplementation should be based on specific deficiency needs

GHK-Cu acts at the extracellular matrix (ECM) level, modulating the balance between matrix metalloproteinases (MMPs) and their inhibitors (TIMPs) to regulate collagen turnover, and stimulating fibroblasts to produce collagen type I, collagen type III, and elastin

The natural aging model was established in randomly selected male and female mice

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