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Various mechanisms, such as glutamate-induced excitotoxicity, mitochondrial dysfunction, neurotoxicity, etc., are associated with the consequences of TBI

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Probiotic treatment prevented MIA-induced increases in IL-6/IL-17a, preserved parvalbumin positive interneurons, normalized GABAergic signaling, and fully rescued social, repetitive, anxiety-and depression-like behaviors in adult offspring (Wang et al., 2019)

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