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By limiting free radical activity, glutathione lowers our oxidative stress that contributes to a loss of collagen and elastin fibres, he continues

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The metabolic reprogramming of CAFs thus represents a novel mechanism of immunotherapy resistance that can be targeted using nanoparticle-based approaches (35)

In cancer, however, DDR exerts a paradoxical influence: while repair deficiencies promote mutagenesis and tumorigenesis, restoration or hyperactivation of DDR pathways in established tumors fosters resistance by enabling efficient repair of therapy-induced DNA damage

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