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The specific differences in their efficacy in reducing EAT volume, improving EAT inflammatory status, and ultimately preventing and treating AF remain uncleara gap that greatly limits clinicians' ability to individualize drug selection based on patient-specific conditions

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Consistent with this observation, a subset of entero-endocrine cells has been shown to co-express GIP and the proglucagon gene, suggesting concomitant secretion of both incretin hormones from such cells [45]

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