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Increases in TMAO levels during CVD/T2D progression (beyond the observed wide natural interindividual variability in TMAO levels) could be the result of disease-related dysbiosis

They can cause adverse effects

They recognize that post-GLP-1 patients may present differently than bariatric patients and can customize techniques accordingly

(2018) reported that GLP-1 induces either an increase or a decrease of spontaneous firing rate in GLP-1R expressing BNST neurons

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