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Signal increases only when membranes are compromised

Tumor necrosis factor-alpha (TNF-), produced by hepatocytes and Kupffer cells, is a key contributor to mitochondrial dysfunction in NAFLD [34]

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All of these phenotypes were markedly reversed by pre-administration of Chlorella vulgaris to mice

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FOXO4-DRI successfully removed senescent chondrocytes, and the results confirmed that removal of senescent cells significantly increased cartilage formation potential and reduced degenerative enzyme production