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Central and peripheral metabolic defects contribute to the pathogenesis of Alzheimers disease: targeting mitochondria for diagnosis and prevention
In a 2013 study, 15 the GSH precursor N -acetyl cysteine (NAC) was administered as a single 60-min intravenous infusion (150 mg/kg) to individuals with PD and healthy controls
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Chiu C, Miller MC, Monahan R, Osgood DP, Stopa EG, Silverberg GD
Therapeutically, this raises two complementary possibilities: (1) SQOR-high tumors may exhibit enhanced mitochondrial ferroptosis resistance through maintained CoQ 10 H 2 and sulfur-based RTAs and (2) tumors with altered sulfur/selenium metabolism may reveal synthetic vulnerabilities when SQOR-linked defense is impaired
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