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Combined research protocols allow investigators to examine whether modulating NAD+ availability alongside mitochondrial redox activity produces measurable effects on cellular energy metabolism endpoints
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Simultaneously, NNMT inhibition preserves SAM the primary methyl-donor for epigenetic histone and DNA methylation reactions which has been shown to influence gene expression programmes governing adipogenesis, lipid storage, and energy expenditure in adipose tissue models
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2 Paul C, Brady DM
In those patients, addressing the underlying metabolic dysfunction first, through dietary intervention, GLP-1 therapy if indicated, or NAD+ support for cellular energy metabolism, is the clinical priority
