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This recent preclinical study showed that the metabolism of racemic ketamine to (2 R ,6 R )-HNK is necessary for the antidepressant effects of ketamine, and that this metabolite itself shows antidepressant-like effects in mice that seem to be independent of the NMDAR
Suzuki H, Kaizuka T, Mizushima N, Noda NN
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This may have set the scene for an adaptive conflict where on one side selection would favour tolerance to a spike in GLP-1 in blood as a result of envenomation and on the other increased potency as a venom component
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