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Homeostatic regulation of protein intake: in search of a mechanism

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C32 and C88 in PfGrx) could not only form an intramolecular disulfide during catalysis or affect the reactivity of the active-site thiolate but also function as a resolving cysteine for trapped Grx(SSR) species (Fig

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One could argue that the higher predisposition to autoimmune disease in women (Cooper and Stroehla, 2003) prompted the sister to react to the subtle white matter damage induced by this mutation in an inflammatory MS-like manner, whereas the brother proceeded along a degenerative path in the absence of the autoimmune overlay

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