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M., Walters, D

In this paper, we explore the potential role of certain amyloidogenic proteins, including amylin, amyloid beta, the prion protein, huntingtin, and -synuclein, in disease processes that result in the accumulation of deposits of protein fibrils, along with lipid membrane components of damaged mitochondria, which we argue may be a mechanism to sequester deuterium in order to reduce the deuterium burden in the tissues

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If there are no or minimal tolerable side effects, you can either maintain the current dose for the next four weeks, or work with our medical team to see if an increased dose is appropriate for you