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By targeting specific microbial populations, EOs can inhibit methanogenic archaea and hyper ammonia-producing bacteria, thereby reducing methane emissions and ammonia concentrations while promoting a fermentation pattern that favors the production of propionate and butyrate, VFAs crucial for energy metabolism [97, 98]

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[10] however, such effects appear to be limited to doses taken over a period longer than 7 days

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These results expanded understanding of Semax's neuroprotective mechanism beyond direct neuronal effects to include modulation of the neuroinflammatory cascade that contributes to secondary brain damage after stroke