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The affinity of rhenium(I) complexes for -amyloid plaques would facilitate the biological evaluation of their technetium(I) analogues as potential radiodiagnostic agents in Alzheimers disease (Sagnou et al., 2011)

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As we age, gene expression changes and the bodys cellular machinery becomes less efficient, leading to a gradual decline in the production of many vital proteins and peptides, including GHK-Cu