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Specifically, in ischemic/high-ROS environments, BACH1 tends to be degraded and acts to suppress angiogenesis by inhibiting the Wnt/-catenin signaling pathway and recruiting HDAC1, among other mechanisms

However, the extent of its long-term safety in humans remains a subject of ongoing investigation

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These reactions include permanent scarring, skin deformities, cysts, severe infections, etc (1)

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