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And they should maintain detailed logs of doses, timing, and observed effects to identify which components are contributing to results
10.1007/s12223-022-00977-2 363 PehA.O'DonnellJ
277,278 Complementing these cellintrinsic models, Farrell extended network adaptation to the tumor microenvironment, arguing that resistance is ecologically driven by crosstalk among cancer cells, CAFs, and macrophages via paracrine signals and exosomes, enabling noncellautonomous drug tolerance
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