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10.1016/j.mce.2007.10.007 34 VroniqueR.PatrickL.StphaneL

Antioxidant Protection Chelates copper(II) ions, reducing free radical damage via superoxide dismutase induction - protective mechanism documented in oxidative stress models

Oxidative Stress & Gene Expression Studies Studies utilizing GHK-Cu report modulation of antioxidant defenses, DNA repair-related transcripts, and cell viability under toxic or oxidative challenge.[4] These data are interpreted as mechanistic evidence for copper-peptidedependent signaling, not as clinical outcomes.

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This disparity is hypothesized to arise from differences in estrogen-mediated neuroprotection, lifestyle factors, and occupational exposure to neurotoxins (Ben-Shlomo et al., 2024) (Figure 2)

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