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[1] [2] As a dual glucose-dependent insulinotropic polypeptide (GIP) and glucagon-like peptide-1 (GLP-1) receptor agonist, tirzepatide works by enhancing insulin secretion, suppressing glucagon release, and slowing gastric emptying

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Directly targeting and depleting total B cells may disrupt other B cell subsets or other immune cell populations, potentially exacerbating OA progression

That tracks with earlier clinical data

Mitochondrial oxidative stress contributes to diastolic dysfunction through impaired mitochondrial dynamics