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This chromatin remodeling restores access to previously silenced genetic regions, including telomerase genes
[1] [2] This triple-receptor mechanism distinguishes it from currently approved agents such as semaglutide (a GLP-1 agonist) and tirzepatide (a dual GIP/GLP-1 agonist)
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The GIP component may also contribute to potentially milder gastrointestinal side effects compared to GLP-1-only medications
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