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Increased FFAs can lead to hepatic lipotoxicity, generation of reactive oxygen, and damage to the mitochondrial membrane (52)

Additionally, exposure to environmental toxins, stress, and certain medical conditions can further deplete glutathione stores in the body

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It acts primarily at the injury site: it upregulates growth hormone receptor expression locally, drives angiogenesis via VEGF, activates the nitric oxide system, and switches on the CREB pathway, the cellular trigger for repair gene expression