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utilized LSKL/PLGA nanoparticles to neutralize TSP-1, significantly inhibiting the TSP-1/CD47 death receptor signaling pathway, thereby reducing mitochondrial membrane potential loss and cytochrome C release, suppressing Bax expression and Caspase-3 activation, and decreasing the TUNEL-positive rate and apoptotic cell count by 50% and 30%, respectively [86]

A block was considered valid if it contained at least one valid Gap and one valid No-Gap trial

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The normal pathways for the synthesis of 2-HG utilize 2-oxoglutarate (-ketoglutarate) or -hydroxybutyrate (GHB) as the substrate