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Based on this mechanism, normalizing excess glutamate levels by metabotropic glutamate group 2/3 receptor agonists has led to potential identification of the first non-monoaminergic target with comparable efficacy as conventional antipsychotic drugs for treating positive and negative symptoms of schizophrenia

GHK peptide inhibits bleomycin-Induced pulmonary fibrosis in mice by suppressing TGF1/Smad-mediated epithelial-to-mesenchymal transition

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