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In cancer, however, DDR exerts a paradoxical influence: while repair deficiencies promote mutagenesis and tumorigenesis, restoration or hyperactivation of DDR pathways in established tumors fosters resistance by enabling efficient repair of therapy-induced DNA damage

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At the optimal cut-off value of 800 ng/ml, NQO1-AS showed 85% sensitivity and 75% specificity, with positive and negative predictive values of 77% and 83%, respectively