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Its ongoing use in laboratory literature reflects research interest only and does not indicate therapeutic, cosmetic, or clinical benefit

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Cystine limitation increased phospho-GCN2 (T899), but not that of PERK (as no visible mobility shift was observed) (Fig

Given their resistance to even prolonged erastin treatment or SLC7A11 KO, these results also suggest that latency III cells have alternative means of meeting cysteine demand, and further suggest that they may utilize non-GPX4 pathways for lipid ROS detoxification (Fig

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