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In conclusion, although there is substantial evidence in animals that numerous selenoproteins (e.g., mGPX4, nGPX4, GPX5, TXNRD3, selenoprotein P) are crucial for normal male reproductive function, and that their deficit (genetic knockdown) leads to reduced or total infertility, the studies in humans are still scarce

This may explain its role in healing damaged tendons

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ALDH2 hampers immune escape in liver hepatocellular carcinoma through ROS/Nrf2-mediated autophagy

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