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This review summarizes current evidence regarding the role of ferroptosis in osteoporosis, with particular focus on its effects on osteoblasts, osteoclasts, and osteocytes, as well as the impact of aging, estrogen deficiency, diabetes, glucocorticoid exposure, and obesity on ferroptosis in osteogenic cells
10.3390/ijms232213828 185 XuX
How it works: IGF-1 LR3 is a modified version of insulin-like growth factor 1, engineered to last 20 to 30 hours in the body instead of 12 minutes
NCOA4, nuclear receptor coactivator 4
Documented in in-vitro assays as a modulator of reactive-oxygen-species pathways and a chelator of free copper, which is itself pro-oxidant
p73 and IGF1R regulate emergence of aggressive cancer stem-like features via miR-885-5p control
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