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Acknowledgements The authors thank Junko Kominami (J-Oil Mills), Noboru Uchiyama, Yusuke Osaka (Shimadzu Co.) and all current and past members of the Hirabayashi Lab for their various contributions to the development and establishment of the FAC protocol
Hyperaldosteronism is associated with impaired vascular reactivity, and it has been shown that aldosterone induces a G6PD-deficient phenotype to impair endothelial function, and aldosterone antagonism or gene transfer of G6PD improves vascular reactivity [9]
Lamprecht M, Bogner S, Steinbauer K, et al
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