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These astrocytes respond to neuronal glutamate via metabotropic glutamate receptors (mGluR), leading to an increase in intracellular Ca 2+

The mechanistic rationale is complementary: GHK-Cu supports collagen synthesis and extracellular matrix remodeling

Selective inhibition of alpha/beta-hydrolase domain 6 attenuates neurodegeneration, alleviates blood brain barrier breakdown, and improves functional recovery in a mouse model of traumatic brain injury

Research supplies (e.g., syringes, bacteriostatic water) are not included

These combined effects create a favorable environment for healing without directly altering structural anatomy

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