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Key structural features include N14E and V17R substitutions (helix stabilisation), 25P/28P/29P proline substitutions (suppression of beta-sheet propensity and amyloid fibril formation, the primary instability mode of native human amylin), a C-terminal proline-to-tyrosine substitution (P37Y) for calcitonin-receptor activity, and N-terminal C20 fatty-diacid acylation via a gamma-Glu linker
However, the drug is not without drawbacks
What they do NOT prove: that these effects translate to meaningful fat loss in humans at any specific dose, or that they occur at comparable magnitude in human visceral versus subcutaneous adipose tissue
It is recommended to wait a week or two before adjusting the dosage if results are not immediate (19)
It is important to monitor your symptoms if you change to oral B12
No IGF-1 elevation
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